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MIF was shown to rescue neurons from oxidative tension induced apoptosis by inhibiting caspase-three activation, and MIF-knockout mice also had much more dead neurons, as well as a better infarct sizing immediately after induction of the experimental stroke [124]. the photographs or other 3rd party website material in this article are included in
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MIF is necessary for the interaction between NLRP3 as well as the intermediate filament protein vimentin, which happens to be vital for NLRP3 activation. additional, we exhibit that MIF interacts with NLRP3, indicating a role for MIF in inflammasome activation impartial of its purpose as a cytokine. These details advance our understanding of how MI